Hyperbaric oxygen therapy (HBOT) improves the circulation of oxygenated blood to wounds to help fight infection,
reduce swelling, and promote healing. The 100 percent oxygen environment and increased atmospheric pressure of HBOT
increase the supply of oxygen to damaged, ischemic tissues. HBOT also reduces edema (excess fluid) in the affected
area, further helping to improve blood flow. Additional benefits occur as the blood supply to damaged tissue improves.
Read the page
Crush Injury, Compartment Syndrome, and other Acute Traumatic Ischemias
in the Undersea and Hyperbaric Medical Society Resource library for more about these conditions; the rationale
for hyperbaric oxygen therapy; and key clinical evidence, outcomes, and success factors.
Hyperbaric oxygen therapy (HBOT) is emerging as a promising treatment for crush injury, compartment syndrome,
and other acute traumatic ischemias. Unfortunately, at present HBOT is “woefully neglected as an adjunct for
managing crush injury and SMCS [skeletal muscle compartment syndrome].” according to the UHMS. This is
particularly regrettable in the case of compartment syndrome because “…in its insipient stages before
a fasciotomy is required…no means to arrest its progression other than hyperbaric oxygen (HBO2) exist.”
[ UHMS, 2015 ]
Traumatic ischemias and
Acute Carbon Monoxide Poisoning,
Central Retinal Artery Occlusion
and other conditions typically seen in an emergency department are reasons why a fully-equipped trauma center should
include hyperbaric oxygen therapy as part of its emergency services portfolio.